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To Live Healthy and Longer, We Should Stop Using Sunflower Oil


  • To Live Healthy and Longer, We Should Stop Using Sunflower Oil

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    Sunflower oil consists of more than half of linoleic acid. The content of fatty acids in sunflower oil (in%): stearic 1.6-4.6, palmitic 3.5-6.4, myristic up to 0.1, arachidonic 0.7-0.9, oleic 24-40, linoleic 46-62, linolenic up to 1. Of the polyunsaturated fatty acids, sunflower oil contains only 1% of omega-3 acids, and Omega-6-unsaturated fatty acids prevail.

    It is in the linoleic acid of sunflower oil that there is a huge potential harm to health!

    Linoleic acid itself is harmless. The problem is that it is very vulnerable to oxygen and iron, which can convert linoleic acid into very toxic substances (HNE4).

    Cardiolipin, a molecule of 4 fatty acids, is found in a mitochondrial membrane and holds mitochondrial complexes through which mitochondria breathe.

    If cardiolipin is damaged, the entire mitochondria cease to function.

    The composition of cardiolipins depends on your diet. An increase in linoleic acid in the diet has an extremely negative effect on cardiolipins.

    Damage to cardiolipins disrupts the electron transport chain. Besides, linoleic acid is converted into dangerous metabolites in cardiolipin. Cardiolipin is only damaged if it is composed of linoleic acid, and this is probably one of the central mechanisms of mitochondrial damage and many diseases.

    Linoleic acid is not essential, so there is no need to use it. Linoleic acid decreases apoptosis (controlled cell death) but increases necrosis (uncontrolled cell death).

    In studies with mice, linoleic acid led to the collapse of mitochondria, then to necrosis and fibrosis in muscle tissues, which then led to myocardial infarction.

    The mice that were poisoned with metabolic toxins to induce diabetes were healthier than the seed oil group.

    Linoleic acid metabolites increase oxidative stress. The antibodies that are produced against these broken cardiolipins correlate with a large number of pathologies: lupus, myocardial infraction, fibromyalgia, chronic fatigue syndrome, atherosclerosis, etc.

    The more linoleic acid in the body, the higher the body's vulnerability to acute respiratory distress syndrome, which is the cause of death in COVID-19.

    Quantitative and qualitative aspects of nutrition affect the deposition of triglycerides in the liver. A diet high in linoleic acids causes liver steatosis.

    Cases have been reported where avoiding linoleic acid has cured fibromyalgia and fatty liver disease.

    There are quite a number of studies (from mice to humans) where a diet with linoleic acid led to weight gain and metabolic syndrome.

    The food industry has been using polyunsaturated fatty acids for animal feed for a long time because they slow down metabolism and less feed is needed.

    For longevity and health, first of all, it is necessary to radically reduce the consumption of linoleic acid and Omega-6 polyunsaturated fatty acids (PUFAs), which are found in sunflower oil.

    It will take about 4 years to replace PUFA in tissues with saturated, more stable acids.


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